Khasm13 said:
Thank God I have not had beef since '91...
^5 khasm...here is more info:
Mad Cow Disease Cover-Up?
Dr. Lester Friedlander, a former USDA vet, had been blowing the whistle on the USDA beef inspection practices before the latest case of BSE (bovine spongiform encephalopathy), was confirmed. Dr. Friedlander said that inspectors are allowed only 15 seconds of inspection and that unhygenic practices are common in the meat industry; practices such as cow-carcass abscesses being hosed off, wrapped up and shipped to the consumer.
The Organic Consumers Association reported last year that hundreds of people are dying in the U.S. each year from CJD (the human counterpart of BSE), and the deaths are being written off as "unexplainable." The disease causes holes in the brains of victims.
Dr. Friedlander also claims that some supervisors were more concerned about falsifying inspection documents than protecting consumers. He claims that on June 9, 2005, a cow in Texas with BSE symptoms was sent straight to the rendering plant without testing.
BSE, commonly known as "mad cow disease," is a fatal, neuro-degenerative disease of cattle, which infects by a mechanism that shocked biologists on its discovery in the late 20th century and appears transmissible to humans. While never having killed cattle on a scale comparable to other dreaded livestock diseases, such as foot-and-mouth and rinderpest, BSE has attracted wide attention because of its apparent transmissibility and lethality to humans, as well as for the nature of the mental decay it causes.
Unlike other kinds of infectious disease that are spread by microbes, the infectious agent in BSE is a specific type of protein. Misshaped ("misfolded") prion proteins carry the disease between individuals and cause deterioration of the brain. BSE is a type of transmissible spongiform encephalopathy (TSE).
TSEs can arise in animals that carry a rare mutant prion
allele, which expresses prions that contort by themselves into the disease-causing shape. Most TSEs, however, occur sporadically - in animals that do not have a prion protein mutation. Transmission can occur when healthy animals consume tainted tissues from other animals with the disease. In the brain these proteins cause native cellular prion protein to deform into the infectious state, which then goes on to deform further prion protein in an exponential cascade. These aggregate to form dense plaque fibers, which lead to the microscopic appearance of "holes" in the brain, degeneration of mental and physical abilities, and ultimately death.
http://www.juiceenewsdaily.com/0605/news/mad_cow.html